EARLY AND LATE COMPLICATIONS OF SURGICAL TREATMENT OF TRAUMATIC INTRACRANIAL HEMATOMAS

Abstract

assessment of early and late complications after surgical treatment of patients with traumatic intracranial
hematomas. Material and methods: 342 patients with traumatic intracranial hematomas were under our control in
Republican Specialized Scientific Practical Medical Center of Neurosurgery. Decompressive craniotomy was performed in
222 (64.9%) patients, osteoplastic craniotomy in 120 (35.1%) patients. Results: Postoperative complications were observed
in 28% of patients with traumatic intracranial hematomas who underwent decompressive craniotomy and 23% after
osteoplastic craniotomy. The most frequent were purulent-inflammatory complications resulted penetrating traumatic
brain injury factors extensive wounds of the soft covers of the skull, reducing the immunological protective properties of the
body, infection Liquorodynamic complications in the form of dysresorptive hydrocephalus (1.4%) and subdural hydroma
(4.1%) were more often observed after decompressive craniotomy. Conclusions: A large number of purulent-inflammatory
complications were caused by a combination of many factors: the presence of penetrating traumatic brain injury in some
patients, extensive wounds of the soft covers of the skull, reduced immunological protective properties of the body in response
to injury, the development of systemic inflammatory syndrome, the addition of nosocomial infection